@article{EschSalberKemperetal.2001,
  author    = {Esch, Thomas and Salber, W. and Kemper, Hans and Staay, F. van der},
  title     = {Der elektromechanische Ventiltrieb - Systembaustein f{\"u}r zuk{\"u}nftige Antriebskonzepte, Teil 2.},
  series = {Motortechnische Zeitschrift (MTZ).},
  volume    = {62},
  journal   = {Motortechnische Zeitschrift (MTZ).},
  number    = {1},
  publisher = {Springer Nature},
  address   = {Basel},
  issn      = {0024-8525},
  doi       = {10.1007/bf03227081},
  pages     = {44 -- 55},
  year      = {2001},
  language  = {de}
}
@article{EschSalberKemperetal.2000,
  author    = {Esch, Thomas and Salber, W. and Kemper, Hans and Staay, F. van der},
  title     = {Der elektromechanische Ventiltrieb - Systembaustein f{\"u}r zuk{\"u}nftige Antriebskonzepte, Teil 1.},
  series = {Motortechnische Zeitschrift (MTZ).},
  volume    = {61},
  journal   = {Motortechnische Zeitschrift (MTZ).},
  number    = {12},
  publisher = {Springer Nature},
  address   = {Basel},
  issn      = {0024-8525},
  doi       = {10.1007/bf03227081},
  pages     = {826 -- 836},
  year      = {2000},
  language  = {de}
}
@article{EschFunkeRoosenetal.2011,
  author    = {Esch, Thomas and Funke, Harald and Roosen, Peter and Jarolimek, Ulrich},
  title     = {Biogene Automobilkraftstoffe in der allgemeinen Luftfahrt},
  series = {Motortechnische Zeitschrift (MTZ).},
  volume    = {72},
  journal   = {Motortechnische Zeitschrift (MTZ).},
  number    = {1},
  publisher = {Springer Nature},
  address   = {Basel},
  isbn      = {0024-8525},
  doi       = {10.1365/s35146-011-0013-7},
  pages     = {54 -- 59},
  year      = {2011},
  language  = {de}
}
@article{Esch2010,
  author    = {Esch, Thomas},
  title     = {Trends in der Nutzfahrzeugantriebstechnik},
  series = {Motortechnische Zeitschrift (MTZ)},
  volume    = {71},
  journal   = {Motortechnische Zeitschrift (MTZ)},
  number    = {10},
  publisher = {Springer Nature},
  address   = {Basel},
  isbn      = {0024-8525},
  doi       = {10.1007/bf03225608},
  pages     = {652 -- 658},
  year      = {2010},
  language  = {de}
}
@article{AtlasBrealeyDharetal.2012,
  author    = {Atlas, Glen and Brealey, David and Dhar, Sunil and Dikta, Gerhard and Singer, Meryvn},
  title     = {Additional hemodynamic measurements with an esophageal Doppler monitor: a preliminary report of compliance, force, kinetic energy, and afterload in the clinical setting},
  series = {Journal of clinical monitoring and computing},
  journal   = {Journal of clinical monitoring and computing},
  number    = {26},
  publisher = {Springer Nature},
  address   = {London},
  isbn      = {1573-2614},
  doi       = {10.1007/s10877-012-9386-5},
  pages     = {473 -- 482},
  year      = {2012},
  abstract  = {The esophageal Doppler monitor (EDM) is a minimally-invasive hemodynamic device which evaluates both cardiac output (CO), and fluid status, by estimating stroke volume (SV) and calculating heart rate (HR). The measurement of these parameters is based upon a continuous and accurate approximation of distal thoracic aortic blood flow. Furthermore, the peak velocity (PV) and mean acceleration (MA), of aortic blood flow at this anatomic location, are also determined by the EDM. The purpose of this preliminary report is to examine additional clinical hemodynamic calculations of: compliance (C), kinetic energy (KE), force (F), and afterload (TSVRi). These data were derived using both velocity-based measurements, provided by the EDM, as well as other contemporaneous physiologic parameters. Data were obtained from anesthetized patients undergoing surgery or who were in a critical care unit. A graphical inspection of these measurements is presented and discussed with respect to each patient's clinical situation. When normalized to each of their initial values, F and KE both consistently demonstrated more discriminative power than either PV or MA. The EDM offers additional applications for hemodynamic monitoring. Further research regarding the accuracy, utility, and limitations of these parameters is therefore indicated.},
  language  = {en}
}
@article{SrivastavaLahiriMaitietal.2014,
  author    = {Srivastava, A. and Lahiri, S. and Maiti, M. and Knolle, F. and Hoyler, Friedrich and Scherer, Ulrich W. and Schnug, E. W.},
  title     = {Study of naturally occurring radioactive material (NORM) in top soil of Punjab State from the North Western part of India},
  series = {Journal of Radioanalytical and Nuclear Chemistry},
  volume    = {2014},
  journal   = {Journal of Radioanalytical and Nuclear Chemistry},
  number    = {302},
  publisher = {Springer Nature},
  address   = {Basel},
  issn      = {1588-2780 (E-Journal); 0022-4081 (Print); 0134-0719 (Print); 0236-5731 (Print); 1417-2097 (Print)},
  doi       = {0.1007/s10967-014-3450-1},
  pages     = {1049 -- 1052},
  year      = {2014},
  language  = {en}
}
@article{RatkeMilowLisinskietal.2014,
  author    = {Ratke, Lorenz and Milow, Barbara and Lisinski, Susanne and Hoepfner, Sandra},
  title     = {On an effect of fine ceramic particles on the structure of aerogels},
  series = {Microgravity science and technology},
  volume    = {26},
  journal   = {Microgravity science and technology},
  publisher = {Springer Nature},
  address   = {Heidelberg},
  issn      = {0938-0108 ; 1875-0494},
  doi       = {10.1007/s12217-014-9380-2},
  pages     = {103 -- 110},
  year      = {2014},
  language  = {en}
}
@article{JordanKruegerWillmesetal.2011,
  author    = {Jordan, Sabine D. and Kr{\"u}ger, Markus and Willmes, Diana M. and Redemann, Nora and Wunderlich, F. Thomas and Br{\"o}nneke, Hella S. and Merkwirth, Carsten and Kashkar, Hamid and Olkkonen, Vesa M. and B{\"o}ttger, Thomas and Braun, Thomas and Seibler, Jost and Br{\"u}ning, Jens C.},
  title     = {Obesity-induced overexpression of miRNA-143 inhibits insulin-stimulated AKT activation and impairs glucose metabolism},
  series = {Nature Cell Biology},
  volume    = {13},
  journal   = {Nature Cell Biology},
  number    = {4},
  publisher = {Nature},
  address   = {New York},
  issn      = {1465-7392},
  doi       = {10.1038/ncb2211},
  pages     = {434 -- 446},
  year      = {2011},
  abstract  = {The contribution of altered post-transcriptional gene silencing to the development of insulin resistance and type 2 diabetes mellitus so far remains elusive. Here, we demonstrate that expression of microRNA (miR)-143 and 145 is upregulated in the liver of genetic and dietary mouse models of obesity. Induced transgenic overexpression of miR-143, but not miR-145, impairs insulin-stimulated AKT activation and glucose homeostasis. Conversely, mice deficient for the miR-143-145 cluster are protected from the development of obesity-associated insulin resistance. Quantitative-mass-spectrometry-based analysis of hepatic protein expression in miR-143-overexpressing mice revealed miR-143-dependent downregulation of oxysterol-binding-protein-related protein (ORP) 8. Reduced ORP8 expression in cultured liver cells impairs the ability of insulin to induce AKT activation, revealing an ORP8-dependent mechanism of AKT regulation. Our experiments provide direct evidence that dysregulated post-transcriptional gene silencing contributes to the development of obesity-induced insulin resistance, and characterize the miR-143-ORP8 pathway as a potential target for the treatment of obesity-associated diabetes.},
  language  = {en}
}
@article{KornfeldBaitzelKoenneretal.2013,
  author    = {Kornfeld, Jan-Wilhelm and Baitzel, Catherina and K{\"o}nner, A. Christine and Nicholls, Hayley T. and Vogt, Merly C. and Herrmanns, Karolin and Scheja, Ludger and Haumaitre, C{\´e}cile and Wolf, Anna M. and Knippschild, Uwe and Seibler, Jost and Cereghini, Silvia and Heeren, Joerg and Stoffel, Markus and Br{\"u}ning, Jens C.},
  title     = {Obesity-induced overexpression of miR-802 impairs glucose metabolism through silencing of Hnf1b},
  series = {Nature},
  volume    = {494},
  journal   = {Nature},
  number    = {7435},
  publisher = {Springer Nature},
  address   = {Cham},
  isbn      = {0028-0836},
  doi       = {10.1038/nature11793},
  pages     = {111 -- 115},
  year      = {2013},
  language  = {en}
}
@article{SchoenrockMuckeltHastermannetal.2024,
  author    = {Schoenrock, Britt and Muckelt, Paul E. and Hastermann, Maria and Albracht, Kirsten and MacGregor, Robert and Martin, David and Gunga, Hans-Christian and Salanova, Michele and Stokes, Maria J. and Warner, Martin B. and Blottner, Dieter},
  title     = {Muscle stiffness indicating mission crew health in space},
  series = {Scientific Reports},
  volume    = {14},
  journal   = {Scientific Reports},
  number    = {Article number: 4196},
  publisher = {Springer Nature},
  address   = {London},
  issn      = {2045-2322},
  doi       = {10.1038/s41598-024-54759-6},
  pages     = {13 Seiten},
  year      = {2024},
  abstract  = {Muscle function is compromised by gravitational unloading in space affecting overall musculoskeletal health. Astronauts perform daily exercise programmes to mitigate these effects but knowing which muscles to target would optimise effectiveness. Accurate inflight assessment to inform exercise programmes is critical due to lack of technologies suitable for spaceflight. Changes in mechanical properties indicate muscle health status and can be measured rapidly and non-invasively using novel technology. A hand-held MyotonPRO device enabled monitoring of muscle health for the first time in spaceflight (> 180 days). Greater/maintained stiffness indicated countermeasures were effective. Tissue stiffness was preserved in the majority of muscles (neck, shoulder, back, thigh) but Tibialis Anterior (foot lever muscle) stiffness decreased inflight vs. preflight (p < 0.0001; mean difference 149 N/m) in all 12 crewmembers. The calf muscles showed opposing effects, Gastrocnemius increasing in stiffness Soleus decreasing. Selective stiffness decrements indicate lack of preservation despite daily inflight countermeasures. This calls for more targeted exercises for lower leg muscles with vital roles as ankle joint stabilizers and in gait. Muscle stiffness is a digital biomarker for risk monitoring during future planetary explorations (Moon, Mars), for healthcare management in challenging environments or clinical disorders in people on Earth, to enable effective tailored exercise programmes.},
  language  = {en}
}