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- Article (1578) (remove)
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Institute
- Fachbereich Medizintechnik und Technomathematik (1578) (remove)
Aneurysmal subarachnoid hemorrhage (aSAH) is associated with early and delayed brain injury due to several underlying and interrelated processes, which include inflammation, oxidative stress, endothelial, and neuronal apoptosis. Treatment with melatonin, a cytoprotective neurohormone with anti-inflammatory, anti-oxidant and anti-apoptotic effects, has been shown to attenuate early brain injury (EBI) and to prevent delayed cerebral vasospasm in experimental aSAH models. Less is known about the role of endogenous melatonin for aSAH outcome and how its production is altered by the pathophysiological cascades initiated during EBI. In the present observational study, we analyzed changes in melatonin levels during the first three weeks after aSAH.
In-beam study of ¹⁴³ Eu
(1988)
Yrast states up to spin 18 have been identified in ¹³²Ce. The energies and spins of the levels suggest the existence of two fairly well-behaved collective bands. The results are compared with those obtained in the rare-earth deformed region.
Side-bands in ¹⁸⁰ Os
(1981)
High spin states in ¹⁸⁸ Au
(1982)
High-spin states in ¹⁸⁰ Os
(1979)
Band structure in ¹⁹⁴ Au
(1979)
Isomeric state in ¹³⁴ La
(1981)
Isomeric state in ¹³⁶ La
(1981)
High-spin states in ¹³³ La
(1980)
High-spin states in ¹³³ La
(1982)
Isomeric states in ¹³⁴ Ba
(1980)
Isomeric states in ¹³⁴ Ba
(1980)
In-beam study of ¹⁴⁴ Gd
(1978)
In-beam study of ¹⁴⁴ Gd
(1977)
In-beam study of ¹⁴⁴ Gd
(1978)
High spin states in ¹³⁶ Ce
(1975)
High spin states in ¹³⁶ Ce
(1975)
Side bands in ¹⁷² Hf
(1978)
New isomeric state in ¹⁴⁵ Eu
(1979)
In-beam study of ¹⁴⁵ Gd
(1979)
New isomeric state in ¹⁴⁴ Eu
(1980)
High spin states in ¹⁹¹ Au
(1975)
High spin states in ¹⁹¹ Au
(1975)
Side bands in ¹⁷² Hf
(1978)